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SH-SY5Y cells which are known to have wild-type p53 have constitutive expression of p53 in the control and why kratom illegal in thailand lower doses groups. The loss of p53 protein was noted as early as 6 hr after MSE treatment. A similar finding was also observed for p21 protein. P21 is one of the main target genes for p53 and both p53 and p21 are well known to have a positive correlation in assisting the cycle arrest by inhibiting the cyclinCdks complex formation (Morgan 2007).
This finding suggests that the mode of the cell death of MIT treated cells is dependant on caspase 3 and 7 activation pathway. There were no significant maeng da kratom powder how to use differences in the subG1 population (apoptosis population) between treated groups (caspase 3 inhibitor kratom tea shelf life caspase 8 inhibitor caspase 9 inhibitor and general caspase inhibitor treated with high dose of MSE) and the control and negative control groups. At this stage it seems that despite having high MIT content in the MSE the high dose MSE treatment in SH-SY5Y cells does not activate caspase enzymes.